IDH3G Polyclonal Antibody, 100ul Immunology Defects in ATG16L1 are a

Defects in ATG16L1 are a cause of susceptibility to inflammatory bowel disease type 10 (IBD10)

an autosomal recessive disorder characterized by hyperammonemia

Tube body designed with both graduation and writing area for easy identification

biologically inert and non-degradable

IDH3G Polyclonal Antibody, 100ul Immunology Defects in ATG16L1 are aIsocitrate dehydrogenases catalyze the oxidative decarboxylation of isocitrate to 2 oxoglutarate. These enzymes belong to two distinct subclasses one of which utilizes NAD(+) as the electron acceptor and the other NADP(+). Five isocitrate dehydrogenases have been reported: three NAD(+) dependent isocitrate dehydrogenases which localize to the mitochondrial matrix and two NADP(+) dependent isocitrate dehydrogenases one of which is mitochondrial and the